The American Heart Association reported that in 2004 alone, myocardial infarction (MI) related mortality rate was as high as 38% among the 1.2 million reported MI cases . After an MI, the self-repair capability of the infarcted heart is limited. As a result, the heart undergoes a remodeling process, characterized by progressive weakening of contractile function. It is believed that increased left ventricular (LV) loading to survival myocytes post-MI activates the apoptotic pathways in the cells, leading to progressive loss of cardiomyocytes.
- Bioengineering Division
Strain Mapping of LV Myocardium and its Correlation With Activation of Apoptotic Molecular Pathways Post Infarction
Wu, C, Li, T, Savai, K, Griffith, BP, & Wu, ZJ. "Strain Mapping of LV Myocardium and its Correlation With Activation of Apoptotic Molecular Pathways Post Infarction." Proceedings of the ASME 2008 Summer Bioengineering Conference. ASME 2008 Summer Bioengineering Conference, Parts A and B. Marco Island, Florida, USA. June 25–29, 2008. pp. 33-34. ASME. https://doi.org/10.1115/SBC2008-193133
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